Recently added item(s)
You have no items in your basket.
Are you confused about multinutrients? Trying to decipher which one would be best for you, or if you actually need one? If chosen well, multinutrients can be an incredibly powerful tool to support many aspects of life, from stress, to immunity, and hormone balance. They can also support different life stages, such as fertility (male and female), pregnancy, and menopause. A carefully formulated complex containing therapeutic doses of nutrients that are easily absorbed and used by the body, is so much more than just a bog standard supplement.
The truth is, most individuals nowadays have an inadequate intake of vitamins and minerals leading to deficiencies.[i] Another key factor to consider is your body’s unique requirements. Think of it as a case of Nutritional Economics - it’s all about supply and demand. If the supply of food is low in nutrient value, or the demand for nutrients is overly high, this can leave us off balance. The reasons for this can include:
The market can appear saturated with multinutrient options, making it confusing and difficult to choose. Not all multinutrients are created equal, so when looking for a multinutrient, choose wisely and make sure to consider the following attributes:
We can really personalise our multinutrient for more targeted supplementation by keeping an eye out on these key nutrients:
When considering a multinutrient, make sure you choose the most effective formula that is easily absorbed and used by the body, with the right ingredients to suit your individual needs, age and lifestyle. A multinutrient is a perfect complementary addition to a healthy diet and lifestyle; a great foundation to build upon, and an insurance policy to guarantee that we are getting everything our body needs to perform optimally.
The brand you can talk to:
Not registered for an account with BioCare®?
[i] Department of Health. (2011). Headline results from Years 1 and 2 (combined) of the rolling programme 2008/9 - 2009/10. National Diet and Nutrition Survey.
[ii] Jones DL et al. REVIEW: Nutrient stripping: the global disparity between food security and soil nutrient stocks. Journal of applied ecology. 2013;50(4):851-862.
[iii] Herrmann W et al. Vitamin B-12 status, particularly holotranscobalamin II and methylmalonic acid concentrations, and hyperhomocysteinemia in vegetarians. Am J Clin Nutr. 2003;78(1):131-6.
[iv] The Vegan Society. Statistics. 2019. https://www.vegansociety.com/news/media/statistics
[v] Melini V, Melini F. Gluten-Free Diet: Gaps and Needs for a Healthier Diet. Nutrients. 2019;11(1):170. Published 2019 Jan 15.
[vi] Weisberg et al. The 1298A-->C polymorphism in methylenetetrahydrofolate reductase (MTHFR): in vitro expression and association with homocysteine. Atherosclerosis, 2001,156:409-15.
[vii] Frosst P et al. A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase. Nat Genet. 1995;10:111-13.
[viii] Oussalah A et al.Association of TCN2 rs1801198 c.776G>C polymorphism with markers of one-carbon metabolism and related diseases: a systematic review and meta-analysis of genetic association studies. Am J Clin Nutr. 2017;106(4):1142–1156.
[ix] Surendran P. et al. An update on vitamin B12-related gene polymorphisms and B12 status. Genes Nutr 2018:13(2).
[x] Tizaoui K et al. Lung. 2014 Association of vitamin D receptor gene polymorphisms with asthma risk: systematic review and updated meta-analysis of case-control studies; 192 (6): 955-65
[xi] Miraglia N et al. Enhanced oral bioavailability of a novel folate salt: comparison with folic acid and a calcium folate salt in a pharmacokinetic study in rats. Minerva Ginecol. 2016;68(2): 99-105.
[xii] AbdRaboh et al. Prevalence of methylenetetrahydrofolate reductase C677T and A1298C polymorphisms in Egyptian patients with type 2 diabetes mellitus. Egyptian Journal of Medical Human Genetics 2013; 14 (1): 87-93.
[xiii] Weisberg et al. The 1298A-->C polymorphism in methylenetetrahydrofolate reductase (MTHFR): in vitro expression and association with homocysteine. Atherosclerosis, 2001,156:409-15.
[xiv] Frosst P et al. A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase. Nat Genet. 1995;10:111-13.
[xv] Henkel et al. Molecular aspects of declining sperm motility in older men. FertilSteril. 2005 Nov;84(5):1430-7.
[xvi] Yamaguchi et al. Zinc is an essential trace element for spermatogenesis. ProcNatlAcadSci U S A. 2009 Jun 30;106(26):10859-64.
[xvii] Rafi et al. Lycopene modulates growth and survival associated genes in prostate cancer. J Nutr Biochem. 2013; 24 (10): 1724-34.
[xviii] Fuhrman et al. Hypocholesterolemic effect of lycopene and beta-carotene is related to suppression of cholesterol synthesis and augmentation of LDL receptor activity in macrophages. Biochem Biophys Res Commun. 1997; 233 (3): 658-62.
[xix] Pekmezci, E., Dundar, C., & Turkoglu, M. (2018). Proprietary Herbal Extract Downregulates the Gene Expression of IL-1α in HaCaT Cells: Possible Implications Against Nonscarring Alopecia. Medical archives (Sarajevo, Bosnia and Herzegovina), 72(2), 136–140. https://doi.org/10.5455/medarh.2018.72.136-140
[xx] Ghorbanibirgani, A., Khalili, A., & Zamani, L. (2013). The efficacy of stinging nettle (urtica dioica) in patients with benign prostatic hyperplasia: a randomized double-blind study in 100 patients. Iranian Red Crescent medical journal, 15(1), 9–10. https://doi.org/10.5812/ircmj.2386
[xxi] Singh RB, Neki NS, Kartikey K, et al. Effect of coenzyme Q10 on risk of atherosclerosis in patients with recent myocardial infarction. Mol Cell Biochem 2003;246:75-82.
[xxii] Kolahdouz Mohammadi R, Hosseinzadeh-Attar MJ, Eshraghian MR, Nakhjavani M, Khorami E, Esteghamati A. The effect of coenzyme Q10 supplementation on metabolic status of type 2 diabetic patients. Minerva Gastroenterol Dietol. 2013 Jun;59(2):231-6.
[xxiii] Safarinejad. The effect of coenzyme Q(10) supplementation on partner pregnancy rate in infertile men with idiopathic oligoasthenoteratozoospermia: an open-label prospective study. Int Urol Nephr. 2012; 44 (3): 689-700.
[xxv] Wyatt et al. Efficacy of vitamin B-6 in the treatment of premenstrual syndrome:systematic review; BMJ 1999;318:1375
[xxvi] Quaranta et al Pilot study of the efficacy and safety of a modified-release magnesium 250 mg tablet (Sincromag) for the treatment of premenstrual syndrome. Clin Drug Investig. 2007;27(1):51-8.
[xxvii] Walker et al. Magnesium supplementation alleviates premenstrual symptoms of fluid retention J Womens Health. 1998 Nov; 7(9):1157-65.
[xxviii] Cho H-J, Yoon I-S. Pharmacokinetic Interactions of Herbs with Cytochrome P450 and P-Glycoprotein. Evidence-based Complementary and Alternative Medicine : eCAM. 2015; 2015:736431
[xxix] Zhu BT, et al. Dietary administration of an extract from rosemary leaves enhances the liver microsomal metabolism of endogenous estrogens and decreases their uterotropic action in CD-1 mice. Carcinogenesis. 1998; 19(10):1821-7
[xxx] Debersac P et al. Induction of cytochrome P450 and/or detoxification enzymes by various extracts of rosemary: description of specific patterns. Food Chem Toxicol. 2001; 39 (9): 907-18
[xxxi] Kass-Annesse. Alternative therapies for menopause. Clin Obstet Gynecol, 43 (1) (2000), pp. 162–183
[xxxii] Tope G et al. Epigallocatechin-3-gallate (EGCG) reduces liver inflammation, oxidative stress and fibrosis in carbon tetrachloride (CCl4)-induced liver injury in mice. Toxicology. 2010; 273(1-3):45-52
[xxxiii] Negri A, Naponelli V, Rizzi F, Bettuzzi S. Molecular Targets of Epigallocatechin-Gallate (EGCG): A Special Focus on Signal Transduction and Cancer. Nutrients. 2018; 10(12):1936
[xxxiv] Kao et al Modulation of endocrine systems and food intake by green tea epigallocatechin gallate. Endocrinology 141: 980–987,2000.
[xxxv] Reid et al. Long-term effects of calcium supplementation on bone loss and fractures in postmenopausal women: a randomized controlled trial. Am J Med 1995;98:331–5.
[xxxvi] Tanaka et al. Cancer Chemoprevention by Carotenoids. Molecules. 2012; 17(3):3202-3242.